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O(6)-Methylguanine-DNA methyltransferase (MGMT), a protein that confers DNA repair and survival advantages to cells, is inactivated in about 50% of human breast cancers. MGMT deficiency has been proposed as a putative pathogen-associated molecular pattern, a unique epigenetic variation specific to tumor cells. In this study, we have generated MGMT knockout clones of the human MCF-7 breast cancer cell line and used them to dissect the HS-3 pathogen-associated molecular pattern recognition system. The HS-3 complement in this system
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